JACC:RAAS抑制剂不会增加COVID-19风险

2020-07-17 MedSci原创 MedSci原创

冠状病毒病-2019(COVID-19)是由严重急性呼吸综合征冠状病毒-2引起的,冠状病毒2通过血管紧张素转换酶2与肾素-血管紧张素-醛固酮系统(RAAS)结合。这种相互作用被认为是RAAS抑制剂治疗

冠状病毒病-2019(COVID-19)是由严重急性呼吸综合征冠状病毒-2引起的,而冠状病毒的细胞表面受体是ACE2。这种相互作用被认为是RAAS抑制剂治疗的潜在危险因素。本研究目的旨在评估RAAS抑制剂是否会影响COVID-19的危险因素。

RASTAVI临床试验是一个正在进行的随机将成功接受经导管主动脉瓣置换术后患者分至雷米普利组和对照组的临床试验。最终共纳入102名患者(50名雷米普利组和52名对照组),平均年龄为82.3 ± 6.1岁,男性占56.9%。其中11名患者被诊断为COVID-19(6名对照组和5名雷米普利组),老年患者(p=0.019)和房颤患者(p=0.066),红细胞压积较低(p=0.084),合并症较多的患者发生COVID-19的风险增加。另外,较高的体重指数是增加死亡率的唯一因素(p=0.039)。

研究结果显示,在老年心血管疾病患者的高危人群中,雷米普利对COVID-19的发病率或严重程度没有影响。该结论支持在COVID-19危重期间维持RAAS抑制剂治疗。

原始出处:

Ignacio J et al.Ramipril in High-Risk Patients With COVID-19.JACC.2020 July.

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    2020-08-13 jklm09
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    2021-05-13 hbwxf
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    2020-09-09 12177770m58暂无昵称

    学习了

    0

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    2020-07-19 wangzheng00

    学习

    0

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    2020-07-19 venlin
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