Circulation:研究发现重度肥胖会降低心衰患者的心肌收缩力

2021-01-06 MedSci原创 MedSci原创

来自约翰霍普金斯医学院的最新研究发现,对于患有这种心衰的患者,重度肥胖会使心肌收缩减弱。

大约一半的心力衰竭患者的心脏收缩能力基本正常,这种综合征被称为射血分数保留的心力衰竭(HFpEF)。来自约翰霍普金斯医学院的最新研究发现,对于患有这种心衰的患者,重度肥胖会使心肌收缩减弱。这项于2020年12月2日发表在《Circulation》杂志上。

左边是正常心脏,右边为HFpEF心脏。有研究发现HFpEF患者心脏的左心室壁较厚、较硬,因此,心室腔中充满足够泵送到全身血液量的空间较小。

“这种心衰的传统药物治疗方式通常会降低心肌收缩强度,以改善心脏舒张。”卡斯说,“我们进一步的研究将会探究这种治疗方法是否需要改变。”

研究人员按照标准的心脏活检程序采集一小块心肌作为HFpEF患者评估的一部分。第一组为从非重度肥胖(平均BMI为30)但患有高血压和心肌肥厚的患者取到的活检样本,第二组为重度肥胖(平均BMI为40)但血压较低且心肌较薄患者。研究人员从这些样本中挑出单个肌肉细胞,通过增加钙离子浓度刺激细胞收缩来研究它们的功能。研究人员发现,当钙离子浓度升高时那些不那么肥胖的人心肌反应正常,而重度肥胖患者的心肌细胞对高钙的力反应降低了40%。第三组为同时患有高血压、心脏增厚,并有重度肥胖患者的心肌细胞,再被给予高钙刺激后,细胞中的力反应与第二组有相似的降低。因此研究员Kass和他的同事认为肥胖是其中的关键因素。

Kass说:“我们还不知道为什么这种力反应会减少,我们正在努力寻找答案,并通过测试新的、不同的药物,来改善心肌收缩能力,为HFpEF和肥胖患者提供个性化治疗。”

原始出处:M. Imran Aslam, Virginia S. Hahn, Vivek Jani, Steven Hsu, Kavita Sharma, David A. Kass. Reduced Right Ventricular Sarcomere Contractility in HFpEF with Severe Obesity. Circulation. 2 Dec 2020

参考资料:https://medicalxpress.com/news/2021-01-obesity-weakens-heart-muscle-patients.html

 

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    2021-07-10 十八线开外美少女

    肥胖降低心肌收缩力

    0

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    2021-01-22 lifefamily

    #肥胖#主要是负荷比较大,减肥用于减少心脏负荷的,有利于#心衰#的治疗

    0

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    2021-01-07 2012iron

    控制体重

    0

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    2021-01-07 ms1000000591710906

    体重指数

    0

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    2021-01-06 Houqiangcn

    减肥

    0

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Circulation:早起、每天睡7-8小时健康,能够大大降低心力衰竭风险

心力衰竭(Heart Failure),简称心衰,是指由于心脏的收缩功能和/或舒张功能发生障碍,不能将静脉回心血量充分排出心脏,导致静脉系统血液淤积,动脉系统血液灌注不足,从而引起心脏循环障碍症候群。

Circulation:2型糖尿病:降低体脂、减小腰围=降低心衰风险!

意向性减肥与2型糖尿病患者的心力衰竭(HF)和动脉粥样硬化心血管疾病风险降低相关。然而,体脂(FM)、瘦体重(LM)和腰围(WC)的基础值和纵向变化对2型糖尿病患者的HF和心肌梗塞(MI)风险的贡献尚

Circ Heart Fail :值得注意!超过半数老年心衰患者出院后服用药物达10种以上

心衰患者常常合并多种疾病,随着治疗方式的不断发展,多种药物治疗也越发普遍,尤其是在老年心衰患者中。一项来自美国的研究显示,超过半数的因心力衰竭住院的老年患者出院后服用的处方药物达到10种或10种以上。

JAHA:循环血管细胞粘附分子-1与心力衰竭的关系

长期随访发现VCAM-1与HF发生率显著相关。这些发现提示内皮活化在促进HF发展特别是HFpEF发展中的潜在作用。减少内皮细胞活化的疗法可能会阻止心血管危险因素发展为临床HF。