Biochemia Medica:维生素K缺乏症II (PIVKA-II)诱导蛋白作为胰腺癌潜在的血清学生物标志物:一项初步研究

2019-08-03 不详 MedSci原创

胃肠道恶性肿瘤患者中,维生素<span lang="EN-US" style="font-size:12.0pt;mso-bidi-font-size:14.0pt;font-family:"Calibri","sans-serif"; mso-fareast-font-family:宋体;mso-bidi-font-family:"Times

肠道恶性肿瘤患者中,维生素K缺乏症(PIVKA-II)诱导蛋白是一种异常的凝血酶原升高。本研究的目的是评价PIVKA-IIPC患者和胰腺良性疾病患者中已建立的胰腺(PC)生物标志物(ca19 -9、癌胚抗原(CEA)ca242)的比较。

我们研究了26PC患者(第1组)和20名良性胰腺疾病患者(第2组)。 通过化学发光酶免疫测定法(CLEIA)在LUMIPULSE G1200Fujirebio-EuropeGentBelgium)上测量PIVKA-IICEA,通过ELSACisBio BioassaysCodoletFrance)和EIAEIA Fujirebio Diagnostics ABGöteborgSweden)测量CA 19-9CA 242。。应用ROC分析评估两组生物标志物的诊断特征。

研究结果显示,第1组和第2组的PIVKA-II中位数和四分位数间距(IQR)分别为1749.0320.2-3921.0)与31.023.0-43.0mAU/mLP <0.001),CA 19-9分别为260.0158.7-272.0)与104.09.0-58.0U/mLP = 0.034);CA 242分别为104.030.2-150.0)与7.24.8-26.0U/mLP <0.050PIVKA-II分别为9.45.3-37.5)与4.51.8-7.0ng/mLP = 0.021)。PIVKA-II, CA 19-9, CA 242, CEAROC曲线下面积分别为0.8695CI0.71-1.00),0.5895CI0.38-0.78),0.7395CI0.54-0.92),0.6495CI0.44-0.85)。

研究表明,在PCPIVKA-II明显高于胰腺良性疾病。与CA 19-9CEACA242相比,PIVKA-II具有较好的诊断性能,对其检测有助于PC管理

原始出处:

Sara Tartaglione ,Irene Pecorella,Protein Induced by Vitamin K Absence II (PIVKA-II) as a potential serological biomarker in pancreatic cancer: a pilot study

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    2020-07-04 jeanqiuqiu
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    2020-04-17 sunylz
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time=2019-08-05, status=1, ipAttribution=)]
    2020-01-23 zhwj
  5. 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  6. 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  7. 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  8. 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