Skip to main page content
U.S. flag

An official website of the United States government

Dot gov

The .gov means it’s official.
Federal government websites often end in .gov or .mil. Before sharing sensitive information, make sure you’re on a federal government site.

Https

The site is secure.
The https:// ensures that you are connecting to the official website and that any information you provide is encrypted and transmitted securely.

Access keys NCBI Homepage MyNCBI Homepage Main Content Main Navigation

Search Page

Filters

My NCBI Filters

Results by year

Table representation of search results timeline featuring number of search results per year.

Year Number of Results
1973 1
1974 2
1976 2
1977 3
1979 1
1980 2
1982 1
1983 1
1984 1
1985 7
1987 3
1988 1
1989 1
1990 1
1991 6
1992 11
1993 7
1994 1
1995 6
1996 8
1997 5
1998 6
1999 2
2000 5
2001 6
2002 12
2003 10
2004 7
2005 10
2006 8
2007 11
2008 8
2009 17
2010 21
2011 22
2012 24
2013 25
2014 24
2015 31
2016 36
2017 36
2018 34
2019 36
2020 41
2021 29
2022 31
2023 47
2024 13

Text availability

Article attribute

Article type

Publication date

Search Results

558 results

Results by year

Filters applied: . Clear all
Page 1
The molecular and metabolic landscape of iron and ferroptosis in cardiovascular disease.
Fang X, Ardehali H, Min J, Wang F. Fang X, et al. Nat Rev Cardiol. 2023 Jan;20(1):7-23. doi: 10.1038/s41569-022-00735-4. Epub 2022 Jul 4. Nat Rev Cardiol. 2023. PMID: 35788564 Free PMC article. Review.
In this Review, we summarize the relationship between the metabolic and molecular pathways of iron signalling and ferroptosis in the context of cardiovascular disease. ...
In this Review, we summarize the relationship between the metabolic and molecular pathways of iron signalling and ferroptosis in the …
Cellular and molecular pathobiology of heart failure with preserved ejection fraction.
Mishra S, Kass DA. Mishra S, et al. Nat Rev Cardiol. 2021 Jun;18(6):400-423. doi: 10.1038/s41569-020-00480-6. Epub 2021 Jan 11. Nat Rev Cardiol. 2021. PMID: 33432192 Free PMC article. Review.
However, new animal models involving both haemodynamic and metabolic disease, and increasing efforts to examine human pathophysiology, are revealing new signalling pathways and potential therapeutic targets. In this Review, we discuss the cellular and molecular path …
However, new animal models involving both haemodynamic and metabolic disease, and increasing efforts to examine human pathophysiology, are r …
Mechanisms and consequences of endothelial cell senescence.
Bloom SI, Islam MT, Lesniewski LA, Donato AJ. Bloom SI, et al. Nat Rev Cardiol. 2023 Jan;20(1):38-51. doi: 10.1038/s41569-022-00739-0. Epub 2022 Jul 19. Nat Rev Cardiol. 2023. PMID: 35853997 Free PMC article. Review.
The vascular endothelium is particularly susceptible to pathogenic stimuli that activate tumour suppressor pathways leading to cellular senescence. We now understand that senescent endothelial cells are highly active, secretory and pro-inflammatory, and have an aber …
The vascular endothelium is particularly susceptible to pathogenic stimuli that activate tumour suppressor pathways leading to cellular …
Mechanisms of physiological and pathological cardiac hypertrophy.
Nakamura M, Sadoshima J. Nakamura M, et al. Nat Rev Cardiol. 2018 Jul;15(7):387-407. doi: 10.1038/s41569-018-0007-y. Nat Rev Cardiol. 2018. PMID: 29674714 Review.
Hypertrophy initially develops as an adaptive response to physiological and pathological stimuli, but pathological hypertrophy generally progresses to heart failure. Each form of hypertrophy is regulated by distinct cellular signalling pathways. In the past decade, a growi …
Hypertrophy initially develops as an adaptive response to physiological and pathological stimuli, but pathological hypertrophy generally pro …
Calcific aortic valve disease: mechanisms, prevention and treatment.
Moncla LM, Briend M, Bossé Y, Mathieu P. Moncla LM, et al. Nat Rev Cardiol. 2023 Aug;20(8):546-559. doi: 10.1038/s41569-023-00845-7. Epub 2023 Feb 24. Nat Rev Cardiol. 2023. PMID: 36829083 Review.
Age and congenital bicuspid aortic valve morphology are important and unalterable risk factors for CAVD, whereas additional risk is conferred by elevated blood pressure and plasma lipoprotein(a) levels and the presence of obesity and diabetes mellitus, which are modifiable factor …
Age and congenital bicuspid aortic valve morphology are important and unalterable risk factors for CAVD, whereas additional risk is conferre …
Macrophage-targeted nanomedicine for the diagnosis and treatment of atherosclerosis.
Chen W, Schilperoort M, Cao Y, Shi J, Tabas I, Tao W. Chen W, et al. Nat Rev Cardiol. 2022 Apr;19(4):228-249. doi: 10.1038/s41569-021-00629-x. Epub 2021 Nov 10. Nat Rev Cardiol. 2022. PMID: 34759324 Free PMC article. Review.
We review the latest progress in nanoparticle-based imaging modalities that can specifically target macrophages. Using novel molecular imaging technology, these modalities enable the identification of advanced atherosclerotic plaques and the assessment of the therapeutic e …
We review the latest progress in nanoparticle-based imaging modalities that can specifically target macrophages. Using novel molecular …
Myocarditis and inflammatory cardiomyopathy: current evidence and future directions.
Tschöpe C, Ammirati E, Bozkurt B, Caforio ALP, Cooper LT, Felix SB, Hare JM, Heidecker B, Heymans S, Hübner N, Kelle S, Klingel K, Maatz H, Parwani AS, Spillmann F, Starling RC, Tsutsui H, Seferovic P, Van Linthout S. Tschöpe C, et al. Nat Rev Cardiol. 2021 Mar;18(3):169-193. doi: 10.1038/s41569-020-00435-x. Epub 2020 Oct 12. Nat Rev Cardiol. 2021. PMID: 33046850 Free PMC article. Review.
Molecular mechanisms of arrhythmogenic cardiomyopathy.
Austin KM, Trembley MA, Chandler SF, Sanders SP, Saffitz JE, Abrams DJ, Pu WT. Austin KM, et al. Nat Rev Cardiol. 2019 Sep;16(9):519-537. doi: 10.1038/s41569-019-0200-7. Nat Rev Cardiol. 2019. PMID: 31028357 Free PMC article. Review.
Mutations in genes that encode components of desmosomes, the adhesive junctions that connect cardiomyocytes, are the predominant cause of arrhythmogenic cardiomyopathy and can be identified in about half of patients with the condition. However, the molecular mechanisms lea …
Mutations in genes that encode components of desmosomes, the adhesive junctions that connect cardiomyocytes, are the predominant cause of ar …
Cellular and mitochondrial mechanisms of atrial fibrillation.
Mason FE, Pronto JRD, Alhussini K, Maack C, Voigt N. Mason FE, et al. Basic Res Cardiol. 2020 Nov 30;115(6):72. doi: 10.1007/s00395-020-00827-7. Basic Res Cardiol. 2020. PMID: 33258071 Free PMC article. Review.
The molecular mechanisms underlying atrial fibrillation (AF), the most common form of arrhythmia, are poorly understood and therefore target-specific treatment options remain an unmet clinical need. ...
The molecular mechanisms underlying atrial fibrillation (AF), the most common form of arrhythmia, are poorly understood and therefore …
558 results