VEGF(165)b, a splice variant of VEGF-A, promotes lung tumor progression and escape from anti-angiogenic therapies through a beta 1 integrin/VEGFR autocrine loop

Boudria, A; Abou Faycal, C; Jia, T; Gout, S; Keramidas, M; Didier, C; Lemaitre, N; Manet, S; Coll, JL; Toffart, AC; Moro-Sibilot, D; Albiges-Rizo, C; Josserand, V; Faurobert, E; Brambilla, C; Brambilla, E; Gazzeri, S; Eymin, B

Eymin, B (reprint author), CNRS, INSERM, Team RNA Splicing Cell Signaling & Response Thera, U1209,UMR 5309, F-38042 Grenoble, France.; Eymin, B (reprint author), Univ Grenoble Alpes, Inst Avancee Biosci, F-38041 Grenoble, France.

ONCOGENE, 2019; 38 (7): 1050

Abstract

Vascular endothelial growth factor-A (VEGF-A) is highly subjected to alternative pre-mRNA splicing that generates several splice variants. The VEGF(xx......

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