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Twist-3 contributions to gamma gamma -> pi(+)pi(-), K+ K- processes in perturbative QCD approach

Asymmetry in PI(3) K activity

First measurement of the CKM angle phi(3) with B-+/--> D(K-S(0)pi(+)pi(-)pi(0)) K-+/- decays

K-3-Rh Protects Against Cerebral Ischemia/Reperfusion Injury by Anti-Apoptotic Effect Through PI3K-Akt

S100A16 COOPERATES WITH DEPDC1 TO PROMOTE THE PROGRESSION AND ANGIOGENESIS OF NEPHROBLASTOMA THROUGH PI3 K/AKT/MTOR PATHWAY

Implications of Phosphoinositide 3-Kinase-Akt (PI3K-Akt) Pathway in the Pathogenesis of Alzheimer's Disease

Liraglutide Protects Nucleus Pulposus Cells Against High-Glucose Induced Apoptosis by Activating PI3K/Akt/ mTOR/Caspase-3 and PI3K/Akt/GSK3 beta/Caspase-3 Signaling Pathways

Search for the suppressed decays B+ -> K+K+pi(-) and B+ -> pi(+)pi K-+(-)

Measurement of J/psi decays into final states 2(pi(+)pi(-))pi(0), K+ K- pi(+)pi(-)pi(0), 2(pi(+)pi(-)) and K+K- pi(+)pi(-)

Amplitude analyses of D-0 -> pi(+)pi(-)pi(+)pi(-)and D-0 -> K+K-pi(+)pi(-)decays

PI(3,4,5)P-3 Engagement Restricts Akt Activity to Cellular Membranes

Phosphatidylinositol-3-kinase (PI3K)/Akt Signaling is Functionally Essential in Myxoid Liposarcoma

Overexpression of 14-3-3 sigma Modulates Cholangiocarcinoma Cell Survival by PI3K/Akt Signaling

Tropomodulin 3 modulates EGFR-PI3K-AKT signaling to drive hepatocellular carcinoma metastasis

Semileptonic transitions: B-(s) -> pi(K); D-s -> K; D -> pi, K; and K -> pi

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