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EGFR-TKIs resistance via EGFR-independent signaling pathways

Discovery of a novel EGFR ligand DPBA that degrades EGFR and suppresses EGFR-positive NSCLC growth

EGFR-AS1 Promotes Bladder Cancer Progression by Upregulating EGFR

Optimized EGFR Blockade Strategies in EGFR Addicted Gastroesophageal Adenocarcinomas

USP22 promotes resistance to EGFR-TKIs by preventing ubiquitination-mediated EGFR degradation in EGFR-mutant

KIF5B-EGFR Fusion: A Novel EGFR Mutation in Lung Adenocarcinoma

Microwave ablation combined with EGFR-TKIs versus only EGFR-TKIs in advanced NSCLC patients with EGFR-sensitive

EGFR Suppression Inhibits the Sphere Formation of MCF7 Cells Overexpressing EGFR

Combination of EGFR-TKIs with chemotherapy versus chemotherapy or EGFR-TKIs alone in advanced NSCLC patientswith EGFR mutation

Diarylheptanoid 35d overcomes EGFR TKI resistance by inducing hsp70-mediated lysosomal degradation ofEGFR in EGFR-mutant lung adenocarcinoma

Expanding the Therapeutic Window of EGFR-Targeted PE24 Immunotoxin for EGFR-Overexpressing Cancers byTailoring the EGFR Binding Affinity

EGFR-TKI Combined with Pemetrexed versus EGFR-TKI Monotherapy in Advanced EGFR-Mutated NSCLC: A Prospective

The EGFR C797S Mutation Confers Resistance to a Novel EGFR Inhibitor CLN-081 to EGFR Exon 20 Insertion

EGFR monoclonal antibody panitumumab inhibits chronic proliferative cholangitis by downregulating EGFR

Comprehensive Analysis of EGFR-Mutant Abundance and Its Effect on Efficacy of EGFR TKIs in Advanced NSCLCwith EGFR Mutations

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