CELL METAB：高脂饮食有助健康长寿？！

2017-09-06 海北 MedSci原创

已有的研究显示，热量限制能够在很多动物模型中延长寿命并推迟年龄相关的疾病。但是至今为止，热量限制有助于增加动物寿命的确切机制仍然受到争议。热量限制会导致机体从碳水化合物代谢转为脂肪代谢。

已有的研究显示，热量限制能够在很多动物模型中延长寿命并推迟年龄相关的疾病。但是至今为止，热量限制有助于增加动物寿命的确切机制仍然受到争议。热量限制会导致机体从碳水化合物代谢转为脂肪代谢。但是我们还需要进一步的研究去证明，是否热量限制相关的脂肪酸氧化和酮生成增加有利于生命周期的延长。相似的，低碳水化合物饮食也能引起从碳水化合物代谢向脂肪代谢的转变。最极端的低碳水化合物饮食，生酮饮食，已被证明能够促进抗炎代谢状态，增加小鼠酮体水平。尽管有这些相似之处，我们还是缺乏动物长期维持这些饮食对于寿命和健康寿命影响的信息。来自加州大学戴维斯分校的研究人员使用低碳水化合物饮食模拟这种代谢转移，并探究这些饮食对小鼠寿命和健康寿命的影响。将12个月龄的C57BL / 6小鼠分配到生酮性低碳水化合物饮食或对照饮食，并在饮食干预1个月或14个月后测试其生理功能，或允许其生长到自然寿命。研究人员发现，和对照组相比，生酮饮食显著增加了小鼠寿命和生存期的中位数。在老年小鼠中，只有采用生酮饮食的小鼠才能保持其生理功能。同时，生酮饮食能够增加蛋白质乙酰化水平，并以组织依赖的方式调节mTORC1信号传导。这项研究表

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2018-05-16 zhaozhouchifen

#Cell#

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2018-04-20 heli0118

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2018-05-27 一闲

#MET#

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2018-08-01 guojianrong

#Meta#

26 0
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2017-09-07 pcw111

#高脂#

28 0
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2017-09-06 ishtar

和柳叶刀的文章异曲同工

76 0
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2017-09-06 明月清辉

谢谢分享.学习了

0 0

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