SLX4IP Antagonizes Promiscuous BLM Activity during ALT Maintenance

Panier, S; Maric, M; Hewitt, G; Mason-Osann, E; Gali, H; Dai, A; Labadorf, A; Guervilly, JH; Ruis, P; Segura-Bayona, S; Belan, O; Marzec, P; Gaillard, PHL; Flynn, RL; Boulton, SJ

Boulton, SJ (reprint author), Francis Crick Inst, 1 Midland Rd, London NW1 1AT, England.

MOLECULAR CELL, 2019; 76 (1): 27

Abstract

Cancer cells acquire unlimited proliferative capacity by either re-expressing telomerase or inducing alternative lengthening of telomeres (ALT), which......

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