Phosphocreatine attenuates doxorubicin-induced cardiotoxicity by inhibiting oxidative stress and activating TAK1 to promote myocardial survival in vivo and in vitro

Wang, C; Hu, L; Guo, S; Yao, Q; Liu, XF; Zhang, B; Meng, XW; Yang, XS

Meng, XW; Yang, XS (corresponding author), Hubei Univ Sci & Technol, Med Res Inst, Hubei Key Lab Diabet & Angiopathy, Xianning 437100, Peoples R China.; Yang, XS (corresponding author), Hubei Univ, Sch Life Sci, State Key Lab Biocatalysis & Enzyme Engn, Wuhan 430062, Peoples R China.

TOXICOLOGY, 2021; 460 ():

Abstract

Myocardial apoptosis and necroptosis are the major etiological factor during doxorubicin (DOX) induced cardiotoxicity, and one of the important reason......

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