Advanced glycation end-products suppress autophagic flux in podocytes by activating mammalian target of rapamycin and inhibiting nuclear translocation of transcription factor EB

Zhao, XC; Chen, YH; Tan, XF; Zhang, L; Zhang, H; Li, ZL; Liu, SX; Li, RZ; Lin, T; Liao, RY; Zhang, QM; Dong, W; Shi, W; Liang, XL

Shi, W; Liang, XL (reprint author), Guangdong Acad Med Sci, Guangdong Gen Hosp, Div Nephrol, 106 Zhongshan 2 Rd, Guangzhou 510080, Guangdong, Peoples R China.

JOURNAL OF PATHOLOGY, 2018; 245 (2): 235

Abstract

Insufficient autophagy in podocytes is related to podocyte injury in diabetic nephropathy (DN). Advanced glycation end-products (AGEs) are major facto......

Full Text Link


Links

期刊讨论 | 中国SCI论文 | 期刊主页 | 投稿经验 | 杂志官网 | 投稿链接 | 作者需知 | PMC链接 | Pubmed全文检索