Inhibition of FLT3 in AML: a focus on sorafenib

Antar, A; Otrock, ZK; El-Cheikh, J; Kharfan-Dabaja, MA; Battipaglia, G; Mahfouz, R; Mohty, M; Bazarbachi, A

Bazarbachi, A (reprint author), Amer Univ, Beirut Med Ctr, Dept Internal Med, Bone Marrow Transplantat Program, POB 113-6044, Beirut 11072020, Lebanon.

BONE MARROW TRANSPLANTATION, 2017; 52 (3): 344

Abstract

FMS-like tyrosine kinase 3 (FLT3) is one of the most commonly mutated genes in AML. FLT3 is mutated in similar to 30% of patients with AML, either by ......

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