原发性输尿管淋巴瘤1例

2019-03-17 高永华 李巧娥 马伟 医学影像学杂志

患者,男,66岁。主因进行性排尿困难2个月入院。2月前无明显原因出现排尿不畅,排尿费力,迟缓断续,射程缩短,尿线变细,终末呈滴沥状,排尿次数明显增加,每日排尿次数大于10次,近日上述症状明显加重。查体:双肾区无隆起,无压痛,无叩击痛。双侧输尿管区未见异常,膀胱无充盈,膀胱区无压痛,阴囊阴茎未见异常。肛诊:前列腺大,质韧,中间沟消失。化验:血常规、肝肾功未见异常,尿常规OB。门诊以“前列腺增生”收

输尿管淋巴瘤分为原发性和继发性两大类,无论是原发性还是继发性,输尿管淋巴瘤都非常罕见。结外脏器淋巴瘤绝大多数为非霍奇金淋巴瘤,霍奇金淋巴瘤极为罕见,免疫组化以B细胞为主,最常见的病理类型为弥漫大B细胞淋巴瘤,T细胞来源较为少见。现将我院收治的1例原发性输尿管淋巴瘤(primary lymphoma of the ureter,PLU)患者临床影像资料结合文献复习报道如下。
 
患者,男,66岁。主因进行性排尿困难2个月入院。2月前无明显原因出现排尿不畅,排尿费力,迟缓断续,射程缩短,尿线变细,终末呈滴沥状,排尿次数明显增加,每日排尿次数大于10次,近日上述症状明显加重。查体:双肾区无隆起,无压痛,无叩击痛。双侧输尿管区未见异常,膀胱无充盈,膀胱区无压痛,阴囊阴茎未见异常。肛诊:前列腺大,质韧,中间沟消失。化验:血常规、肝肾功未见异常,尿常规OB。门诊以“前列腺增生”收入院。
 
检查方法
 
用Siemens Definition AS+128层螺旋CT机,采用连续容积扫描模式,先行全尿路平扫,然后行肾皮质期、肾实质期增强扫描及排泄期全尿路扫描,扫描完毕数据传至工作站,运用Syngo软件进行三维后处理。主要扫描参数:120kV,102~280有效mAs,层厚8mm,层间距8mm的容积扫描,然后1mm层厚、0.5层间距重建,重建卷积核B30f,扫描范围从胸11椎体下缘至耻骨联合下缘。
 
结果
 
超声检查
 
前列腺肥大,右肾积水,右侧输尿管全程扩张并下段近膀胱入口处不规则混合回声,建议进一步检查。
 
膀胱镜检查
 
右侧输尿管口处可见大小约60mm×50mm×40mm乳头样肿物,开口显示不清;前列腺增大,以两侧叶为主。
 
CT扫描
 
右肾体积缩小,皮质变薄,肾盂积水扩张,右侧输尿管全程增粗,直径约13~18mm,走行自然无僵直,管壁间断性不均匀增厚,局部管腔狭窄,输尿管远端可见不规则软组织密度影突入膀胱,大小约30mm×40mm,密度均匀,边界欠清,增强扫描右肾未见明显强化,输尿管病变轻度强化,延迟扫描右侧肾盂输尿管未见对比剂充盈;前列腺41mm×53mm。CT诊断右侧输尿管占位性病变(淋巴瘤可能)伴肾积水,右肾功能减低;前列腺肥大(图1~7)。
 
手术所见
 
入院第5天在全麻下行右侧输尿管及膀胱部分切除术、右侧输尿管再植术。术中探查见右侧输尿管口处有一直径约4 cm的肿瘤,基底宽,质硬。沿肿瘤边缘周围2 cm处切除正常全层膀胱壁,包括肿瘤及右侧输尿管口。术中见输尿管下段8 cm及膀胱右侧壁受肿瘤侵犯,与盆底粘连紧密,输尿管与周围组织粘连紧密,游离右侧输尿管,于右侧正常膀胱壁处切一小口,行右侧输尿管膀胱再植术(术后右侧输尿管口未见喷尿),缝合膀胱壁及浆肌层,留置尿管及皮下引流管,依次缝合腹壁各层。
 
病理结果
 
右侧输尿管B细胞淋巴瘤(图8)。
 


图1~8为同一患者,CT显示右肾体积缩小,皮质变薄,肾盂积水扩张,右侧输尿管全程增粗,走行自然无僵直,管壁间断性不均匀增厚,局部管腔狭窄,输尿管远端可见不规则软组织密度影突入膀胱,密度均匀,边界欠清,增强扫描右肾未见明显强化,输尿管病变轻度强化,延迟扫描右侧肾盂输尿管未见对比剂充盈。图8右侧输尿管膀胱入口处B细胞淋巴瘤(HE×200)
 
原始出处:

高永华,李巧娥,马伟,吕丽琴,杨喜银,郭苗.原发性输尿管淋巴瘤1例并文献复习[J].医学影像学杂志,2018(01):159-162.

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    2019-03-18 txqjm

    谢谢了,学习

    0

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    2019-03-18 深海的鱼

    继续努力学习

    0

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    2019-03-18 飛歌

    学习了很有用不错

    0

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    2019-03-17 天地飞扬

    了解一下,谢谢分享!

    0

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